you may notice problems with the display of certain parts of an article in other ereaders. person with osa may experience loud snoring, oxygen desaturation, frequent arousal, and disruption of sleep. the global prevalence of osa varies from 0.3% to 5.1% in general population.  patients of asthma appear to have an increased risk for osa than general population. the first study that examines asthma and osa was a case report by hudgel and shucard in 1979.
 in this landmark study, the incidence of osa over 4 years in patients with self-reported asthma was 27%, compared with 16% without asthma.  further, it is to be noted that osa and asthma are often complicated by shared comorbidities and potential for multidirectional casual pathways, i.e., obstruction, inflammation, obesity, gastroesophageal reflux disorder, and rhinitis. there is accumulating evidence to suggest that osa is associated with the poor asthma control, more nocturnal symptoms, and frequent exacerbations in asthma patients. osa is largely overlooked in patients having uncontrolled asthma. patients with poor asthma control despite optimal recommended management need to be screened for possible osa so as to reduce treatment cost, morbidity, and improved quality of life in such patients.
clarifying the nature of the relationship between osas and asthma is a critical area with important therapeutic implications. obstructive sleep apnea syndrome has been shown to be associated with inflammation of both the upper and lower respiratory tracts. one proposed mechanism for airway inflammation in osas is the mechanical stress exerted on the mucosa by the high negative pressures transmitted against a closed airway passage as a result of the strong inspiratory effort produced by snoring and obstructive apneas.
the relationship between elevated leptin levels in osas patients and airway hyperresponsiveness and inflammation could be an important causal link between the morbidities of osas and asthma. another cause of the high incidence of osas in asthmatic patients may be the reduction of airway cross-sectional area and upper airway patency. recent data suggest that osas is an independent risk factor for asthma exacerbations153 and that osas symptoms are more common in asthmatic patients than in the general population.
[6,7] epidemiological studies demonstrate that asthma patients more frequently report snoring, excessive daytime sleepiness, and apnea. a recent population- several studies have confirmed that asthmatic patients are more prone to develop osas symptoms than are members of the general population. the common asthmatic asthma and obstructive sleep apnea (osa) are long-term respiratory disorders that affect breathing. asthma causes inflammation and narrowing, .
people with asthma are at increased risk of developing sleep apnea. being overweight, smoking and alcohol use are also risk factors. sleep apnea also tends to run in families. in some cases, it results from body structure u2013 having a small upper airway, for example, or a recessed chin, small jaw or large neck size. obstructive sleep apnea (osa) and asthma are closely related. this could be due to mere coexistence, shared risk factors, or distinct over the full study period, the team concluded that asthma patients faced an almost 40 percent greater risk for sleep apnea than asthma-free osa risk relates to poor overall asthma control during daytime and nighttime. additionally, osa treatment leads to improved asthma symptoms,, .
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