in this case study, we describe a 35-year-old woman with pulmonary hypertension that was secondary to thyrotoxicosis who presented with exertional dyspnea. we demonstrated the reversibility of pulmonary hypertension and its symptoms following treatment with radioactive iodine. next, to investigate the cause of the shortness of breath and pulmonary hypertension, the patient was referred to the pulmonary hypertension clinic.
in one study, most of the patients with pulmonary hypertension due to hyperthyroidism were asymptomatic.15 conversely, some studies have found that the prevalence of thyroid disease is significantly higher in patients with severe pulmonary hypertension. some studies have evaluated the role of methimazole, compared with surgery, in the regulation of pulmonary vascular resistance in patients with hyperthyroidism and pah. the increase in pap usually reverses after treatment of thyroid disease; therefore, some patients may have symptomatic pulmonary hypertension that can be resolved by treatment with radioactive iodine.
a 33-year-old woman presented to an outside facility with increasing shortness of breath, orthopnea, paroxysmal nocturnal dyspnea and increasing lower limb edema — findings suggestive of biventricular heart failure. the patient was diuresed and started on beta-adrenergic blockade and 10 mg of methimazole three times a day. the patient’s presentation with decompensated heart failure, preceding gastrointestinal complaints, and tachycardia was highly suggestive of thyroid storm (burch and wartofsky point scale 40). the patient’s clinical picture was most consistent with cardiogenic shock in the setting of thyroid storm of autoimmune origin, and possible postpartum cardiomyopathy.
following total thyroidectomy, the patient improved and was able to be weaned off vasopressor support and she was extubated. unfortunately, her cardiac function remained suboptimal (ejection fraction of 15 percent), and therefore a left ventricular assist device was placed to bridge to cardiac transplantation. the patient was able to be discharged home with an lvad, awaiting heart transplantation. diagnostic criteria, clinical features, and incidence of thyroid storm based on nationwide surveys.
dyspnea is recognized to be an important feature in patients with hyperthyroidism at rest and during exercise. however, its etiology is not well-understood. palpitations and shortness of breath improved, and both her thyroid-stimulating hormone and t4 levels decreased. dyspnea is recognized to be an important feature in patients with hyperthyroidism at rest and during exercise. however, its etiology is not well-understood., hyperthyroidism symptoms, hyperthyroidism symptoms, how does hyperthyroidism cause shortness of breath, hypothyroidism, why does hypothyroidism cause shortness of breath.
both hypothyroidism and hyperthyroidism cause respiratory muscle weakness and decrease pulmonary function. hypothyroidism reduces respiratory drive and can cause obstructive sleep apnea or pleural effusion, while hyperthyroidism increases respiratory drive and can cause dyspnea on exertion. a number of breathing and/or lung issues can result from hyperthyroidism, including: dyspnea (difficult or labored breathing) dyspnea on exertion is a common complaint in hyperthyroidism, and this thyroid dysfunction has been implicated as a primary cause of impaired effort summary breathlessness is a common complaint in patients with hyperthyroidism, thyroid. prior to therapy, reduced quadriceps muscle strength, a 33-year-old woman presented to an outside facility with increasing shortness of breath, orthopnea, paroxysmal nocturnal dyspnea and, hyperthyroidism symptoms in females, shortness of breath thyroid medication.
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