osa is a condition that requires management and the disease can be treated by using cpap therapy. a four-year longitudinal study  of overweight and obese american adults demonstrates that change in weight is directly proportionate to sleep disordered breathing (sdb). an increase in the level of ghrelin and a decrease in the level of leptin is also noted in chronic short sleepers [15,16]. another study shows that increased levels of the leptin hormone are present in both obese and obstructive sleep apnea patients, with the leptin level being directly proportional to the severity of the syndrome . the prevalence of osa in the general population is 3 to 7% for men and 2 to 5% for women . in the sleep heart health study, a multi-ethnic cohort was studied to explore the symptoms of sdb in relation to race and ethnicity.
weight loss is the key factor in the treatment of osa. the concentration of the enzyme heme oxygenase is increased in patients with severe osa and morbid obesity; bariatric surgery decreases this enzyme concentration. after bariatric surgery, bleeding, ulceration, fistula formation, and stenosis of the anastomosis can occur; all these complications can be repaired endoscopically or in an open procedure . the prevalence of osa is increasing globally due to the growing occurrence of obesity in society. the gold standard of treatment is cpap therapy and ensuring that patients adhere to the therapy. solving this issue is not only the responsibility of an individual and a healthcare worker, but it should be properly addressed by government officials to improve the health of individuals and make an overall healthier society.
in obstructive sleep apnea (osa), the most common type of sleep apnea, disruptive breathing occurs because of a narrow or blocked upper airway. several health conditions increase the likelihood of developing sleep apnea, but osa is most common in people who are overweight or obese. sleep apnea can also deplete people of the energy they need to maintain a healthy body weight. the cyclic rising and falling blood oxygen levels can cause inflammation, which in turn may lead to atherosclerosis (a build-up of plaque in the blood vessels) which is associated with heart attacks, stroke, and high blood pressure.
as a result, sleep apnea is associated with the following heart, lung, and metabolic problems, among others: osa frequently coexists in people with obesity hypoventilation syndrome (ohs). unfortunately, while weight loss can provide meaningful improvements in osa, it usually does not lead to a complete cure, and many sleep apnea patients need additional therapies. regardless of technique, osa improvement is proportional to the amount of weight lost. dr. debanto is a medical doctor and gastroenterologist with 20 years of experience in obesity management and related issues such as sleep apnea.
there is a linear correlation between obesity and osa. in obese people, fat deposits in the upper respiratory tract narrow the airway; there is excess body weight contributes to sleep apnea by causing increased pressure on upper airways, leading to collapse and decreased neuromuscular in adults, the most common cause of obstructive sleep apnea is excess weight and obesity, which is associated with the soft tissue of the, .
several cross-sectional studies have consistently found an association between increased body weight and the risk of osa. significant sleep apnea is present in u224840% of obese individuals,17 and u224870% of osa patients are obese. medical conditions linked to osa include hypertension, insulin resistance, visceral fat deposition, and systemic inflammation. sleep apnea is believed to affect 25% of the adult population and as high as 45% of individuals with obesity. but being overweight is still a high risk factor for the development of obstructive sleep apnea. on one hand, carrying the extra weight can lead to breathing obesity has long been recognised as the most important reversible risk factor for obstructive sleep apnoea (osa). analyses from the wisconsin sleep cohort, .
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