the transition from acute to chronic insomnia is presaged by baseline differences in sleep architecture that have, in the past, been ascribed to major depression, either as heritable traits or as acquired traits from prior episodes of depression. as such, the initiation of insomnia and its acute phase is thought to occur in response to life-event stress. if the subject met the criteria for normal sleep or acute insomnia, they were asked if they had a history of acute or chronic insomnia. there were no differences in attrition between normal sleepers and those with acute insomnia between being asked to take part in the psg assessment and completion of the study (x2(1) = 1.04, p = 0.31). finally, those who reported normal sleep at baseline and continued to report normal sleep at 1 month and 3 months (i.e., they had to report being satisfied with their sleep and no current difficulty in initiating sleep, maintaining sleep, or waking too early in the morning) were classified as normal sleepers.
then, data on sleep status (normal sleeper, individual with chronic insomnia, or natural remitter) and levels of depression derived from the 3-month follow-up survey are reported. in terms of comparisons on sleep architecture, differences were evident within rem latency (p < 0.01) and percentage of n3 (p < 0.006): the chronic insomnia group exhibited a reduced rem latency as compared to normal sleepers (p < 0.05) and natural remitters (p < 0.05) and those with chronic insomnia exhibited reduced n3 compared to normal sleepers (p < 0.05). the primary aims of this study were to determine whether differences existed in terms of life events, perceived stress, mood, and sleep, both subjectively and objectively, between those with acute insomnia and normal sleepers. this is the first study of its kind in terms of examining objective and subjective sleep in naturally occurring acute insomnia and examining baseline characteristics with a short temporal resolution. the present study sought, for the first time, to characterize stress, sleep, and mood during acute insomnia and to determine whether any of the factors present at the onset of insomnia differentiated those who would transit to chronic insomnia from those who would remit.
events like a job loss or the death of a loved one often cause some sleepless nights. your doctor might call it acute insomnia as long as it at some point, many adults experience short-term (acute) insomnia, which lasts for days or weeks. it’s usually the result of stress or a sleep onset insomnia describes difficulty falling asleep at the beginning of the night, or in the case of shift workers, whenever they, how to cure insomnia in 12 minutes, causes of insomnia in females, causes of insomnia in females, insomnia test, insomnia treatment.
sleep onset insomnia or acute onset may be traced to various triggers such as sudden changes in lifestyle, the onset of certain short-term stressors, or poor sleep hygiene. this may include a loud or noisy environment, intake of coffee or a large meal shortly before bedtime, or lack of adequate exercise. psychological or psychiatric issues are the most common causes. these include stress, anxiety, or depression. according to a 2009 study, people with chronic onset insomnia often have another sleep disorder, such as restless leg syndrome or periodic limb movement disorder. the evolution to chronic insomnia is thought to be largely, if not wholly, mediated by perpetuating factors that are behavioral in nature, relating to how the acute insomnia lasts for less than three months and is often related in time to an identifiable cause. insomnia is present when there is more specifically, individuals that take ≥ 30 minutes to fall asleep or who are awake for periods of this magnitude during the night are, chronic insomnia, types of insomnia.
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